We present a case of nonarteritic anterior ischemic optic neuropathy (NA-AION) with rapidly progressive bilateralization and unclear etiology. We report the case of a 58-year-old man with no specific past medical history who was consulted for a sudden, non-painful, unilateral visual defect in his left eye after waking from sleep. His visual acuity was 10/10 in both eyes. Anterior segment examination was unremarkable in both eyes, but a positive Marcus Gunn signal in the left eye. Fundoscopic examination, OCT examination, fluorescein and indocyanine green angiography (ICG) confirmed papilledema in the left eye. The octopus visual field (OVF) showed an inferior altitudinal scotoma. Thus, after ruling out other diagnoses, a diagnosis of NAION was made in the left eye, the patient was consulted for the same symptoms in the right eye Vision was limited to counting fingers at 4 meters in the right eye and 10/10 in the left eye Anterior segment examination was unremarkable in both eyes, but OCT examination of the fundus (RAPD) in the right eye Fluorescein and indocyanine green (ICG) angiography confirmed papilledema in the right eye OVF showed tunnel vision in both eyes blood tests and imagery were negative The diagnosis of non-arteritic AION in the right eye was retained. A therapeutic trial was performed; the patient received a corticosteroid bolus of 1g /day for 3 days, followed by oral treatment 1 month later, visual acuity was 0.4/10 in the right eye and 10/10 in the left eye. Fundus examination and OCT scan showed regression of the edema in the right eye and papillary atrophy in the left eye with tunnel vision in both eyes with OVF